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More and more evidence shows that the ubiquitination of RTK is the key to its lysosomal degradation and recruitment of the ubiquitin protein ligase Cbl family is required for ligand-induced degradation of many RTKs

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Nonetheless, the present analysis is designed to complement, not replace, these long-term analyses, which are pertinent for fully capturing the long-term complications associated with diabetes, which can be influenced by changes in HbA1c and additional secondary parameters [2, 3, 7]