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Prader-Willi syndrome: a review of clinical, genetic, and endocrine findings
Soedamah-Muthu, S
These tumors originate from melanocytes distributed throughout the oral mucosa, particularly in the hard palate, maxillary alveolar ridge, and gingiva, where they exist in a chronically inflamed microenvironment lacking protective stratum corneum [11]
Ethics declarations Acknowledgements The authors thank all study participants and the laboratory technicians L
They screened six differentially expressed ferroptosis-related genes (IL6, ANGPTL7, CDKN1A, AKR1C3, NOX4, and VLDLR) and constructed a random forest model based on these genes, which demonstrated strong diagnostic performance in the validation dataset, highlighting the close relationship between ferroptosis and immunity in atherosclerosis pathogenesis (61)
