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In pancreatic ductal adenocarcinoma, lactate accumulation fuels the enrichment of histone H3 lysine-18 lactylation (H3K18la) at the promoters of TTK and BUB1B, generating a glycolysisH3K18laTTK/BUB1B positive-feedback circuit that persistently augments glucose uptake and lactate production, thereby reinforcing aerobic glycolysis [90]

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Importantly, circulating DPP4 is augmented in obese and type 2 diabetic subjects, and it may represent a molecular link between obesity and vascular dysfunction

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