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Kazandjian D, Blumenthal GM, Chen HY, He K, Patel M, Justice R, Keegan P, Pazdur R

Extracellular deposition of -amyloid (A plaques), intraneuronal tau accumulation, inflammation (activated astrocytes and microglia), and neuronal loss are all consistent pathological features of the disease (Porquet et al., It is well established that A peptides existed in both fibrillary and non-fibrillar forms (Poduslo and Howell, 42 and A 40 are both neurotoxic using both human and murine neuronal cell cultures

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