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The central clinical idea: when mitochondrial ATP production falls and oxidative stress rises, the brain may lose the energy and antioxidant reserve needed to regulate neurotransmission, maintain cell membranes, recycle methionine, produce SAM, clear adenosine and homocysteine, build glutathione and tolerate medications or supplements

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[69] found that high concentrations of calcium and phosphate were able to inhibit the SLC7A11/GSH/GPX4 pathway in rat vascular smooth muscle cells, thereby inducing ferroptosis

Pergialiotis, V

Ascorbate can decrease the repair and restoration of protein functions [174]

Glutathione is critical to our health because its a major antioxidant