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The role of GLP-1R in AD is reflected mainly in the following aspects: it can decrease A deposition and inhibit tau protein hyperphosphorylation, reduce neuroinflammation and oxidative stress (OS) ( 2.1 GLP-1R and A deposition A is a protein fragment produced by the cleavage of amyloid precursor protein (APP) by a series of enzymes and is one of the key factors in AD research (Sambamurti et al., 2002

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The functional medicine approach views it as a condition that involves multiple body systems, including the immune, digestive, and nervous systems

36 Results from the Semaglutide and Walking Capacity in People with Symptomatic Peripheral Artery Disease and T2DM (STRIDE) trial also found that semaglutide 1.0 mg once weekly is associated with an increase in walking distance in individuals with symptomatic peripheral artery disease and T2DM

134979-01-4, CH2COOH-PEG2-NH2.HCl References: [1] Tan, Q., Akindehin, S