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Neuropathological investigations in the substantia nigra pars compacta of Parkinsons disease patients demonstrate that deficient PGC-1 expression compromises mitochondrial biogenesis and disrupts the coordinated transcriptional activation of antioxidant stress-responsive gene networks, thereby precipitating dopaminergic neuronal degeneration (Piccinin et al., 2021)

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Neurons pretreated with Dihexa demonstrate significantly enhanced survival when exposed to reactive oxygen species, excitotoxic glutamate concentrations, or metabolic impairment compared to untreated controls

El uso puede generar molestias y efectos secundarios tanto locales como generales