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In this Review, we evaluate the translational evidence supporting GLP-1RAs as candidate disease-modifying therapies for NDDs, describe mechanistic pathways linking metabolic dysfunction to neuronal vulnerability, and outline remaining challenges and research priorities in this rapidly evolving field

For replication analysis, genetic associations from the FinnGen dataset were used

Given that the risk factors for all-cause mortality among the four poor outcomes were relatively well established, to verify the methodological error of the Cox analysis for the results in Table 4 results, we performed univariable and multivariable Cox proportional hazards analyses including traditional, AAV-specific, and inflammation-related risk factors along with GPX-3 concentration at diagnosis for all-cause mortality during follow-up (Murray et al., 2013

Non-Small Cell Lung Cancer - Resectable Disease

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